Hypoparathyroidism results in decreased PTH production leading to:

Hypoparathyroidism is characterized by inadequate production of parathyroid hormone (PTH), which plays a crucial role in regulating calcium levels in the bloodstream. PTH typically acts to increase serum calcium levels through various mechanisms: it stimulates the release of calcium from bones, promotes calcium reabsorption in the kidneys, and increases intestinal absorption of calcium via its influence on vitamin D metabolism.

When there is decreased PTH production, the opposite effect occurs, leading to hypocalcemia, or low levels of calcium in the blood. This condition can result in various symptoms, including muscle cramps, spasms, and neurological disturbances due to the critical role calcium plays in muscle contraction and nerve signaling.

The other conditions presented in the choices do not arise from decreased PTH levels. Hypercalcemia refers to high calcium levels, hypertension relates to high blood pressure and is not directly influenced by PTH levels in this context, and hyperglycemia is associated with elevated blood glucose levels, typically linked to insulin dysregulation, not calcium management. Therefore, hypocalcemia is the direct and correct consequence of hypoparathyroidism due to the reduced levels of PTH.